Socionic Profiles of Vasopressin

And now we move on to obtaining socionic profiles of vasopressin.
To do this, we will use not its psychological effects (which are not always unambiguous), but its well-studied physiological manifestations.
These include reduced daily water intake, reduced daily urination, and faster cessation of bleeding (the latter caused by the fact that vasopressin has a vasoconstrictive effect on damaged capillaries – further enhanced by platelet aggregation under the influence of serotonin released from them at wound sites).

VP-1. Low daily water intake (cluster of high vasopressin, with additional influence from reduced dopamine and reduced histamine)

High vasopressin is the main factor suppressing daily water consumption. Acting on vasopressin receptors in kidney cells, vasopressin reduces urination, thereby helping to retain fluid in the body and reducing the need to drink. But there are two more factors also influencing daily water intake. One is high dopamine. It promotes increased excretion of table salt (natriuresis) through urine, which causes increased salt intake, which in turn causes increased thirst and increases water consumption. We are able to account for this factor by constructing a salt consumption profile. We will adjust the VP-1 cluster profiles for this dopamine-related salt consumption factor to neutralize dopamine’s influence.

Elevated histamine also partly increases water consumption. The point here is that high histamine creates a tryptophan deficiency in the CNS – tryptophan being a precursor of serotonin. Therefore, the subject with high histamine unconsciously tries to increase the amount of water in the body (drinks a lot), since water indirectly helps to replenish tryptophan reserves and raise serotonin. This factor is only the third in significance and is difficult to account for, so in analyzing cluster VP-1 we will ignore the role of histamine and not make adjustments for it.

1. During the day I drink rarely and rarely go to the bathroom for urination. 0.899
2. I usually drink water, tea, or mineral water only during meals, and not much; I drink rarely and little outside of mealtime. 0.840
3. I drink little and rarely; my body does not need a large amount of water. 0.501
4. I know that I can go a day without drinking water and do fine without it, not feeling strong thirst. 0.458
5. There are days when I only drink a couple of glasses of liquid in 24 hours, no more. 0.239

6. I’m a “water guzzler,” I have a habit of drinking a lot of water (tea, mineral water, or other liquid) every day. -0.945
7. I drink a lot of water daily. -0.903
8. I drink a lot of water daily. -0.892
9. I have a habit of drinking a lot of water (tea, mineral water, or other liquid) daily. -0.876
10. I often feel like having a sip or at least rinsing my mouth – it feels dry in the mouth. -0.864
11. I usually need to drink something while eating, otherwise it’s hard to eat – my mouth is often too dry. -0.661
12. I drink a lot and often – water leaves my body quickly. -0.429

High salt intake and thirst (elevated dopamine, enhanced natriuresis) – a correction scale for isolating the role of vasopressin in daily water intake

1. I often crave something salty. 0.653
2. Tomato and vegetable salads in cafes and canteens usually seem under-salted to me. 0.623
3. Almost daily I like to snack on something salty at lunch (salted fish, pickled vegetables, etc.). 0.620
4. I often feel very thirsty. 0.604
5. Food often seems under-salted to me – probably because I generally like salty things. 0.521
6. I like all sorts of pickles and often eat quite a lot of them (herring, pickles, sauerkraut, pickled mushrooms, etc.). 0.442
7. I’m drawn to salty foods. 0.042

8. I consume salt daily with food, probably in smaller amounts than others – I rarely eat salty foods. -0.752

If we remove the correlation of VP-1 with daily salt intake (i.e., remove the additional influence of peripheral dopamine on the cluster), we get the corrected VP-1 cluster – in the third attached illustration it is labeled:

Low daily water intake (isolated vasopressin contribution, with neutralization of dopamine's stimulating influence on daily water consumption)

VP-2. Slowed urination (elevated vasopressin)

This cluster is most directly associated with vasopressin concentration in the body's biological fluids. However, its precise assessment via questionnaires is the least reliable (which is why we rely on several different clusters to obtain an integrated vasopressin profile).

1. It’s true that I usually go to the bathroom for urination rarely – on average no more than a couple of times a day. 0.860
2. During the day I drink rarely and rarely go to the bathroom for urination. 0.843
3. If I drink a couple of glasses of water, usually I won’t need to urinate for 4–5 hours or more (i.e., water is excreted from the body with some delay). 0.769

4. Often when anxious I begin to feel strong urges on the bladder. -0.778
5. I often experience frequent urination when in a mood (usually in an excited state). -0.583
6. I experience frequent urination – I often feel the urge to go "number one". -0.480

VP-3. Rapid cessation of bleeding (vasoconstrictive influence of vasopressin in blood lymph on damaged capillaries; possibly also affected by elevated peripheral serotonin in blood platelets and several other specialized peptides)

1. My bleeding stops quickly when I get cuts. 0.939
2. It’s true that my bleeding is usually light and stops quickly (in cuts or abrasions, in women during menstruation, etc.). 0.878
3. Compared to other people, my bleeding stops quickly. 0.876

4. Even small cuts take a long time to stop bleeding. -0.860
5. There have been cases of nosebleeds lasting a long time, not stopping. -0.721
6. Any bruise almost always ends up as a colorful bruise on me. -0.711

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By averaging the profiles of the three physiological action clusters of vasopressin (VP-1 adjusted for salt intake, VP-2, and VP-3), we finally obtain the average vasopressin profiles across its three marker clusters. The corresponding final socionic profile of vasopressin can be considered definitive – it is shown in the final attached illustration.

As we can see from this illustration, vasopressin increases the functions Di, Se, and Ti (especially Di), and decreases a number of other functions (primarily Ne and Fe).
At the level of socionic traits, vasopressin significantly increases declatimity, and consequently also somewhat increases statics, logic, sensorics, and decisiveness.

The results are quite consistent with what is known from literary sources about the psychological effects of vasopressin – as a predominantly “male” neurohormone that promotes logic, spatial orientation, and in some cases, aggressive behaviors.

According to the obtained socionic profiles, the highest vasopressin levels should on average be observed in the SLE type, and the lowest – in the EIE type.